How to treat pulmonary edema? Will it become cancerous?

(1) Etiological treatment is very important for the prognosis of pulmonary edema, which can alleviate or correct the disorder of fluid exchange inside and outside pulmonary vessels. If the infusion speed is too fast, stop or slow down immediately. Uremia patients can be treated by dialysis. People who induce infection should apply appropriate antibiotics immediately. Poison gas inhalers should leave the scene immediately and give antidote. Those who take too much anesthetic should immediately lavage their stomachs and take countermeasures.

(2) subcutaneous or intravenous injection of morphine at a dose of 5 ~ 10 mg can relieve anxiety, reduce the resistance of peripheral blood vessels through the inhibition of central sexual intercourse, and transfer blood from pulmonary circulation to systemic circulation. It can also relax respiratory smooth muscle and improve ventilation. It has the best effect on cardiogenic pulmonary edema, but it is forbidden for shock, respiratory depression and chronic obstructive pulmonary disease complicated with pulmonary edema.

(3) Intravenous injection of furosemide (40 ~ 100 mg) or butachlor 1mg can rapidly induce diuresis, reduce circulating blood volume, increase plasma colloid osmotic pressure and reduce the amount of fluid filtered by microvessels. In addition, intravenous injection of furosemide can dilate veins, reduce venous reflux, and even reduce pulmonary edema before diuretic effect. But it is not suitable for people with insufficient blood volume.

(4) Patients with pulmonary edema usually need to inhale higher concentration of oxygen to improve hypoxemia, and it is best to use a mask to give oxygen. 75 ~ 95% alcohol or 10% silica gel is built in the humidifier to help eliminate foam. For patients with refractory hypoxemia, oxygen can be given through mask or artificial airway, which is helpful to increase interstitial hydrostatic pressure, reduce cardiac output, reduce microvascular hydrostatic pressure and reduce fluid filtration outside blood vessels, but it is forbidden for those with insufficient cardiac output.

(5) Intravenous drip of sodium nitroprusside (15 ~ 30μ g/min) can dilate arterioles and venules. Alpha blockers can block the vasoconstriction of catecholamine, histamine and serotonin, and dilate arterioles and venules in lung and systemic circulation. Both of them can reduce the load before and after the heart, reduce the blood flow of pulmonary circulation and the hydrostatic pressure of microvascular, and then reduce pulmonary edema. Commonly used benzylamine oxazoline 0.2 ~ 1 mg/min or benzylbenzyl 0.5 ~ 1 mg/kg intravenous drip. However, attention should be paid to adjusting the number of drops and supplementing the blood volume to keep the arterial blood pressure within the normal range.

(6) Cardiotonic drugs are mainly suitable for pulmonary edema induced by rapid atrial fibrillation or atrial flutter. For those who have not used digitalis drugs within two weeks, 0,25 mg or 0.4 ~ 0.8mg of verbascoside C can be dissolved in glucose for slow intravenous injection.

(7) Intravenous injection of 0.25 g aminophylline can effectively dilate bronchi, improve myocardial contractility, increase renal blood flow and excrete sodium. But pay attention to the injection speed to prevent adverse effects on the heart.

(8) There are differences in the therapeutic value of adrenal glucocorticoids on pulmonary edema. Studies have shown that it can reduce inflammatory reaction, reduce microvascular permeability, promote the synthesis of surfactant, enhance myocardial contractility, reduce peripheral vascular resistance and stabilize lysosomal membrane. Can be used for treating high altitude pulmonary edema, toxic pulmonary edema and myocarditis complicated with pulmonary edema. Dexamethasone 20 ~ 40 mg/d or hydrocortisone 400 ~ 800 mg/d, usually by intravenous injection for 2 ~ 3 days.

(9) Reduce the blood volume of pulmonary circulation. When the patient is sitting, his legs droop or his limbs are tied with venous tourniquets in turn, and one limb is relaxed for 5 minutes every 20 minutes, the blood volume of venous reflux can be reduced. It is suitable for infusion overload or cardiogenic pulmonary edema, and is forbidden for patients with shock and anemia.

The interstitial phase of pulmonary edema can show the effect on hemodynamics. The increase of interstitial hydrostatic pressure can compress nearby microvessels, increase pulmonary circulation resistance and increase pulmonary artery pressure. Hypoxia and acidosis can also directly contract pulmonary blood vessels, further worsen hemodynamics, aggravate right heart load and cause cardiac insufficiency. If not corrected in time, he may die of heart failure and arrhythmia.

Can't wait for cancer.